ANGIOFIBROMA NASOFARING JUVENILE PDF

Nasopharyngeal angiofibroma is a histologically benign but locally aggressive vascular tumor that grows in the back of the nasal cavity. It most commonly affects adolescent males and may grow into fissures of the. Nasoangiofibroma youth is a highly vascularized tumor almost exclusively male adolescents presentation. % represents % of head and. Angiofibroma nasofaring juvenile – Free download as PDF File .pdf), Text File . txt) or read online for free.

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However, most authors agree that JNAs arise from the posterior choanal tissues in the region of the sphenopalatine foramen. Extensive bony destruction is usually not a feature, but rather bone is remodelled or resorbed. Unfortunately, this is difficult due to innocuous presenting symptoms. InFriedberg called it juvenile angiofibroma. Steroid hormone receptor expression in nasopharyngeal angiofibromas. Click here for patient related inquiries.

Endoscopic approach is an excellent tool in primary and recurrent JNA, it allows visualisation and precise removal of the lesion. Several reports suggest that JNA regresses over time. Thus, early diagnosis, accurate staging and adequate treatment are essential in the management of this lesion.

Juvenile nasopharyngeal angiofibroma

It is an aggressive neoplasm and shows a propensity for destructive local spread often extending to the base of the skull and into the cranium. MRI is superior to CT for detecting soft tissue extension of the tumor intracranially. Immunohistochemical and electron microscopical characterization of stromal cells in nasopharyngeal angiofibromas.

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The histologic origin of JNA involves vascular endothelial cells or fibroblasts. Irradiation may be an option if surgery is not possible or only incomplete resection achieved 2,4,6. The exact site of origin is contentious as these masses usually qngiofibroma when they have reached considerable size. You can also scroll through angiofibromq with your mouse wheel or the keyboard arrow keys.

Histopathological examination revealed a proliferative connective tissue stroma interspersed with a thick vascular network. The blood vessels are slit-like or dilated, organized in clusters and are of different calibers. Page views in Current debate involves the hamartoma and vascular malformation theories. Androgen receptor, juvenile angiofiborma, nasopharyngeal angiofibroma. Intracranial extension can however occur.

jufenile CT images show a heterodense mass that is centered in the sphenopalatine foramen. Finally, numerous inflammatory cells such as mast cells and T-lymphocytes are seen. No encapsulation was noted [ Figure 4 ]. The infratemporal fossa approach for nasopharyngeal tumors.

Thus, early diagnosis, accurate staging, and adequate treatment are essential in the management of this lesion. Please review our privacy policy.

Antral sign or Holman-Miller sign forward bowing of posterior wall of maxilla is pathognomic of angiofibroma. Conflicts of interest There are no conflicts of interest. Well circumscribed but unencapsulated polypoid fibrous mass, bleeds severely on manipulation and biopsy, may occlude nares Spongy cut surface.

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Juvenile nasopharyngeal angiofibroma

Mortality is not associated with nasopharyngeal angiofibroma. The origin and development of JNA is not fully understood. Case 6 Case 6. JNA is classified as Type I when the tumor is restricted to the nasal cavity and the nasopharynx without bone destruction, Type II when the tumor invades the pterygomaxillary fossa and maxillary, sphenoidal and ethmoid sinuses with bone destruction, Type III when the tumor invades the infratemporal fossa, the orbit, and the parasellar region but remains lateral to the cavernous sinus and Type IV when the tumor invades the cavernous sinus, the optic chiasma and the pituitary fossa.

Strayer; Emanuel Rubin JNA is benign but locally destructive. Continuous growth involves the sphenoidal sinus, nasal fossa and middle turbinate, pterygomaxillary fossa and the posterior wall of the maxillary sinus as seen in the present case.

Diagnosis is arrived at by clinical examination, radiography, nasal endoscopy and specialized imaging techniques such as CT scan and MRI. Invasion of the intracranial region may lead to cranial nerve palsy.